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Folate and nucleic acid metabolism The figure highlights that riboflavin in the form of flavin adenine dinucleotide (FAD) is a required cofactor for the enzyme methylenetetrahydrofolate reductase (MTHFR), which reduces 5,10-methylenetetrahydrofolate to 5-methyltetrahydrofolate

It promotes the formation of new blood vessels, which improves nutrient delivery to healing tissues

Depletion of cellular antioxidant defences, such as GSH, results in accumulation of ROS, loss of mitochondria function and thus induction of cell death

On the other hand, excessive iron levels within cells after FeCl 3 injection in the rat cerebral cortex can aberrantly activate the mitochondrial oxidative phosphorylation pathway